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Activation of these receptors triggers two complementary processes: it accelerates lipolysis (the breakdown of stored triglycerides into free fatty acids for energy) while simultaneously inhibiting lipogenesis (the conversion of glucose and other substrates into new fat stores)

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The higher dose in the second half of the cycle is designed to reinforce the collagen synthesis, sirtuin activation, and anti-inflammatory pathways initiated in Phase 1

doi: 10.1002/rmb2.12303 Summary Keywords endometrial, endometrial dysfunction, endometrial disease, obesity, oxidative stress Citation Wang Y, Chen Z, Wang R, Song A and Zhang Y (2026) Obesity and oxidative stress: potential mechanisms in endometrial disorders