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demonstrated that the co-administration of Diane-35 and metformin resulted in decreased androgen receptor expression, increased GLUT4 expression, and modulation of cellular autophagy initiation by inhibiting IGF-1, ultimately leading to the suppression of EC development mediated by the mTORC pathway (Liu Y

Similarly, Inzucchi et al [27] believed that the evidence of GLP-1 receptor agonist cardiovascular protection was still limited, and the cardiovascular system benefits related to GLP-1 receptor agonists may be independent of its glucose, lipid, or energy metabolism effects

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